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FPWR Blog

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Another ghrelin knockout mouse

Below is an abstract of a paper just published in the Proceedings of the National Academy of Sciences (PNAS) discussing a new mouse model in which they have completely deleted the ghrelin gene. (you'll recall that individuals with PWS frequently have...
May 24, 2004 Read More

Topics: Research

Orexin promotes wakefulness in a mouse model

You may remember that orexin is a small protein (peptide) released in the brain, which was originally identified because it stimulates feeding. In addition to a role in feeding/energy regulation, it is also critical in wakefulness. People with narcol...
April 1, 2004 Read More

Topics: Research

Leptin replacement in humans

We all know leptin regulates appetite. It is a hormone, released by fat cells, that there is plenty of fat around -- essentially a 'stop eating' signal produced by fat cells and received in the brain. When leptin was first identified, people thought ...
March 31, 2004 Read More

Topics: Research

Ghrelin receptor knockout mouse

This is an article that is very likely to generate a lot of discussion in the field. This study is trying to dissect the importance of ghrelin in regulating growth and appetite. They made a mouse that is missing the receptor for ghrelin (GHSR).
March 31, 2004 Read More

Topics: Research

Clinical trial of modafinil in children

Here is a recent study (link to abstract below) looking at the use of modafinil, a wake-promoting drug, in children with excessive daytime sleepiness (in this study, primarily narcolepsy).
January 26, 2004 Read More

Topics: Research

Genes in the BP1/BP2 interval on chromosome 15 identified

New research has identified genes affected in Prader-Willi patients by deletion. Deletions account for approximately 70% of PWS cases. There are two different sizes of deletion, with one type encompassing more genes than the other.
October 2, 2003 Read More

Topics: Research

About FPWR

The Foundation for Prader-Willi Research (federal tax id 31-1763110) is a nonprofit corporation with federal tax exempt status as a public charity under section 501(c)(3).

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The mission of FPWR is to eliminate the challenges of Prader-Willi syndrome through the advancement of research and therapeutic development.

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